semiwet electrotransfer system (Bio-Rad)
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semiwet electrotransfer system
Semiwet Electrotransfer System, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 96/100, based on 1730 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nitrocellulose+membranes+by+electrotransfer/Nitrocellulose+Membrane%2E+0%2E45+%CE%9Cm/pm40644788-115-12-15
Average 96 stars, based on 1730 article reviews
Semiwet Electrotransfer System, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 96/100, based on 1730 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nitrocellulose+membranes+by+electrotransfer/Nitrocellulose+Membrane%2E+0%2E45+%CE%9Cm/pm40644788-115-12-15
Average 96 stars, based on 1730 article reviews
semiwet electrotransfer system - by Bioz Stars,
2026-09
96/100 stars
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Electrophoresis:Article Title: Expression of islet inducible nitric oxide synthase and inhibition of glucose-stimulated insulin release after long-term lipid infusion in the rat is counteracted by PACAP27. Article Snippet: .. After electrophoresis, proteins were transferred to Article Title: Role of nitric oxide synthase isoforms in glucose-stimulated insulin release. Article Snippet: Henningsson, Ragnar, Albert Salehi, and Ingmar Lundquist.. Role of nitric oxide synthase isoforms in glucosestimulated insulin release.. Am J Physiol Cell Physiol 283: C296–C304, 2002. Article Title: Defective glucose-stimulated insulin release in the diabetic Goto-Kakizaki (GK) rat coincides with reduced activity of the islet carbon monoxide signaling pathway. Article Snippet: .. After electrophoresis, proteins were transferred to Article Title: Carbon monoxide stimulates insulin release and propagates Ca2+ signals between pancreatic beta-cells. Article Snippet: Lundquist, Ingmar, Per Alm, Albert Salehi, Ragnar Henningsson, Eva Grapengiesser, and Bo Hellman.. Carbon monoxide stimulates insulin release and propagates Ca2 signals between pancreatic -cells.. Am J Physiol Endocrinol Metab 285: E1055–E1063, 2003; 10.1152/ ajpendo.00498.2002.—A key question for understanding the mechanisms of pulsatile insulin release is how the underlying -cell oscillations of the cytoplasmic Ca2 concentration ([Ca2 ]i) are synchronized within and among the islets in the pancreas. Article Title: Evaluation of islet heme oxygenase-CO and nitric oxide synthase-NO pathways during acute endotoxemia Article Snippet: Henningsson, Ragnar, Per Alm, and Ingmar Lundquist.. Evaluation of islet heme oxygenase-CO and nitric oxide synthase-NO pathways during acute endotoxemia.. Am J Physiol Cell Physiol 280: C1242–C1254, 2001.—We investigated, by a combined in vivo and in vitro approach, the temporal changes of islet nitric oxide synthase (NOS)-derived nitric oxide (NO) and heme oxygenase (HO)-derived carbon monoxide (CO) production in relation to insulin and glucagon secretion during acute endotoxemia induced by lipopolysaccharide (LPS) in mice. Article Title: Total Parenteral Nutrition Modulates Hormone Release by Stimulating Expression and Activity of Inducible Nitric Oxide Synthase in Rat Pancreatic Islets Article Snippet: 97 The expression and activities of constitutive nitric oxide synthase (cNOS) and inducible nitric oxide synthase (iNOS) in relation to insulin and glucagon secretory mechanisms were investigated in islets isolated from rats subjected to total parenteral nutrition (TPN) for 10 d. TPN is known to result in significantly increased levels of plasma lipids during the infusion time.. In comparison with islets from freely fed control rats, islets taken from TPN rats at d 10 displayed a marked decrease in glucose-stimulated insulin release (4.65 ± 0.45 ng/[islet.h] vs 10.25 ± 0.65 for controls) (p < 0.001) accompanied by a strong iNOS activity (18.3 ± 1.1 pmol of NO/[min.mg of protein]) and a modestly reduced cNOS activity (11.3 ± 3.2 pmol of NO/[min.mg of protein] vs 17.7 ± 1.7 for controls) (p < 0.01).. Similarly, Western blots showed the expression of iNOS protein as well as a significant reduction in cNOS protein in islets from TPN-treated rats. Electrotransfer:Article Title: Expression of islet inducible nitric oxide synthase and inhibition of glucose-stimulated insulin release after long-term lipid infusion in the rat is counteracted by PACAP27. Article Snippet: .. After electrophoresis, proteins were transferred to Article Title: Role of nitric oxide synthase isoforms in glucose-stimulated insulin release. Article Snippet: Henningsson, Ragnar, Albert Salehi, and Ingmar Lundquist.. Role of nitric oxide synthase isoforms in glucosestimulated insulin release.. Am J Physiol Cell Physiol 283: C296–C304, 2002. Article Title: Defective glucose-stimulated insulin release in the diabetic Goto-Kakizaki (GK) rat coincides with reduced activity of the islet carbon monoxide signaling pathway. Article Snippet: .. After electrophoresis, proteins were transferred to Article Title: Carbon monoxide stimulates insulin release and propagates Ca2+ signals between pancreatic beta-cells. Article Snippet: Lundquist, Ingmar, Per Alm, Albert Salehi, Ragnar Henningsson, Eva Grapengiesser, and Bo Hellman.. Carbon monoxide stimulates insulin release and propagates Ca2 signals between pancreatic -cells.. Am J Physiol Endocrinol Metab 285: E1055–E1063, 2003; 10.1152/ ajpendo.00498.2002.—A key question for understanding the mechanisms of pulsatile insulin release is how the underlying -cell oscillations of the cytoplasmic Ca2 concentration ([Ca2 ]i) are synchronized within and among the islets in the pancreas. Article Title: Evaluation of islet heme oxygenase-CO and nitric oxide synthase-NO pathways during acute endotoxemia Article Snippet: Henningsson, Ragnar, Per Alm, and Ingmar Lundquist.. Evaluation of islet heme oxygenase-CO and nitric oxide synthase-NO pathways during acute endotoxemia.. Am J Physiol Cell Physiol 280: C1242–C1254, 2001.—We investigated, by a combined in vivo and in vitro approach, the temporal changes of islet nitric oxide synthase (NOS)-derived nitric oxide (NO) and heme oxygenase (HO)-derived carbon monoxide (CO) production in relation to insulin and glucagon secretion during acute endotoxemia induced by lipopolysaccharide (LPS) in mice. Article Title: Total Parenteral Nutrition Modulates Hormone Release by Stimulating Expression and Activity of Inducible Nitric Oxide Synthase in Rat Pancreatic Islets Article Snippet: 97 The expression and activities of constitutive nitric oxide synthase (cNOS) and inducible nitric oxide synthase (iNOS) in relation to insulin and glucagon secretory mechanisms were investigated in islets isolated from rats subjected to total parenteral nutrition (TPN) for 10 d. TPN is known to result in significantly increased levels of plasma lipids during the infusion time.. In comparison with islets from freely fed control rats, islets taken from TPN rats at d 10 displayed a marked decrease in glucose-stimulated insulin release (4.65 ± 0.45 ng/[islet.h] vs 10.25 ± 0.65 for controls) (p < 0.001) accompanied by a strong iNOS activity (18.3 ± 1.1 pmol of NO/[min.mg of protein]) and a modestly reduced cNOS activity (11.3 ± 3.2 pmol of NO/[min.mg of protein] vs 17.7 ± 1.7 for controls) (p < 0.01).. Similarly, Western blots showed the expression of iNOS protein as well as a significant reduction in cNOS protein in islets from TPN-treated rats. SDS Page:Article Title: Recombinant core proteins of Japanese encephalitis virus as activators of the innate immune response. Article Snippet: Nitric oxide (NO) has been shown to suppress Japanese encephalitis virus (JEV) RNA synthesis, viral protein accumulation, and virus release from infected cells.. In this article, the potential viral structural proteins as the activators of NO product were studied at the molecular level.. First, the genomic region encoding the JEV structural proteins was cloned into a prokaryotic expression vector pET for high-level expression. |